systemic lupus erythematosus (SLE) to inform diagnostic and therapeutic decisions. Describe and explain the key events in the. • pathogenesis. PDF | Systemic lupus erythematosus (SLE) is a chronic autoimmune disease affecting Novel observations have provided an improved understanding of the . systemic lupus erythematosus (SLE) over the past 50 years. This change results from earlier .. Lyn: lck/yes-related novel; SLE: Systemic lupus erythematosus.

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Journal of the American Society Nephrology 15 2 : — Lupus 15 2 : 71 — Autoimmune Diseases 1 — Waldman M, Madaio M Pathogenic autoantibodies in lupus nephritis. Lupus 14 1 : 19 — Philadelphia, Lippincott William and Wilkins.

Autoimmunity Reviews 7 4 : — Journal of Immunology Research 1 — 8. Journal of Biomedicine and Biotechnology 1 — 7. Kidney International Supplements 3 1.

Clinical Chemistry 51 9 : — Dahlan S Besar sampel dan cara pengambilan sampel dalam penelitian kedokteran dan kesehatan. Jakarta, Penerbit Salemba Medika.

This variation probably impacted on past epidemiological estimates and diminished the comparability of earlier cSLE studies [ 4 ]. The authors suggest an upper age limit for the diagnosis of cSLE at 18 years based on the published literature and issues pertaining to the provision of medical care. The same review attempts to achieve more uniformity in terminology when referring to children and adolescents with onset of SLE prior to adulthood [ 4 ].

SLE is also more common among non-White populations.

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Changes in the ethnic and racial composition of some countries, such as the United States, Canada, and several European countries, may partly explain the rising incidence and prevalence of SLE in some geographic regions. The importance of sociodemographic factors and changed environmental factors for the development of SLE is supported by the highest SLE prevalence in the South of the United States and in high-poverty areas [ 5 , 6 ].

Among the Likewise, the annual incidence of cSLE is 2. With respect to lupus nephritis, the annual incidence and prevalence rates of lupus nephritis are 0.

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This compares with annual incidence and prevalence rates of lupus nephritis at 6. Updates on pathogenesis The pathogenesis of SLE remains a vibrant area of research [ 8 ].

Type 1 interferons are thought to be associated with the increased incidence of SLE in early adulthood. Indeed, about one-half of genetic variants associated with SLE to date can be linked to type 1 interferon production or signaling.

There is increasing evidence that miRNAs are involved in the differential expression of SLE-associated genes [ 11 , 12 ].

Controversies in lupus: nervous system involvement. Ann Rheum Dis.

Systemic lupus erythematosus. Diabetes insipidus.

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Text book of internal medicine. Review article: the antiphospolipid syndrome.

N Engl J Med ; Setiyohadi B. The management of systemic lupus erythematosus. Complete papers of the rheumatology scientific meeting.

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Jakarta: Indonesian Rheumatism Association; B cell-intrinsic TLR7 signaling is essential for the development of spontaneous germinal centers. Vasopressin cell antibodies and central diabetes insipidus in a patient with systemic lupus erythematosus and dermatomyositis.

A T-box transcription factor T-bet also contributes to pathogenic autoantibody production [ 35 , 37 ]. As per this meta-analysis fever, thrombocytopenia, mucocutaneous involvement, urinary casts, seizures, and hemolytic anemia are all more commonly encountered in cSLE than aSLE, with ORs ranging from 1.


Oleh sebab itu, gaya bicaranya dibuat sesastra mungkin. Autoimmun Rev.

Buku ajar ilmu penyakit dalam. Diam Belum Tentu Emas

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